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Biological Activity for DSLET
δ Opioid receptor agonist.
Technical Data for DSLET
(Modifications: Ser-2 = D-Ser)
|Storage||Desiccate at -20°C|
The technical data provided above is for guidance only. For batch specific data refer to the Certificate of Analysis.
Tocris products are intended for laboratory research use only, unless stated otherwise.
References for DSLET
References are publications that support the biological activity of the product.
Hiller et al (1996) Autoradiographic comparison of [3H]DPDPE and [3H]DSLET binding: evidence for distinct δ1 and δ2 opioid receptor populations in rat brain. Brain Res. 719 85 PMID: 8782867
Yukhananov et al (1994) Opiate withdrawal intensity correlates with the presence of DSLET high-affinity binding. Pharmacol.Biochem.Behav. 49 1109 PMID: 7886084
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Keywords: DSLET, DSLET supplier, Selective, δ-opioid, delta-opioid, agonist, peptide, DOP, Receptors, OP1, [D-Ser2,, Leu5,, Thr6]-enkephalin, Delta, Opioid, 1170, Tocris Bioscience
Citations for DSLET
Citations are publications that use Tocris products.
Currently there are no citations for DSLET.
Reviews for DSLET
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Literature in this Area
Tocris offers the following scientific literature in this area to showcase our products. We invite you to request* your copy today!
*Please note that Tocris will only send literature to established scientific business / institute addresses.
Peptides Involved in Appetite Modulation Scientific Review
Written by Sonia Tucci, Lynsay Kobelis and Tim Kirkham, this review provides a synopsis of the increasing number of peptides that have been implicated in appetite regulation and energy homeostasis; putative roles of the major peptides are outlined and compounds available from Tocris are listed.
The key feature of drug addiction is the inability to stop using a drug despite clear evidence of harm. This poster describes the brain circuits associated with addiction, and provides an overview of the main classes of addictive drugs and the neurotransmitter systems that they target.
Peripheral sensitization is the reduction in the threshold of excitability of sensory neurons that results in an augmented response to a given external stimulus. This poster outlines the excitatory and inhibitory signaling pathways involved in modulation of peripheral sensitization. The role of ion channels, GPCRs, neurotrophins, and cytokines in sensory neurons are also described.