CHEB

Discontinued Product

CHEB (Cat. No. 0311) has been withdrawn from sale for commercial reasons.
Description: Convulsant barbiturate
Chemical Name: 5-(2'-Cyclohexylidenylethyl)-5-ethylbarbituric acid
Datasheet
Citations
Reviews
Literature (7)

Biological Activity for CHEB

Convulsant barbiturate.

Technical Data for CHEB

M. Wt 264.32
Storage Store at RT
CAS Number 22173-64-4

The technical data provided above is for guidance only. For batch specific data refer to the Certificate of Analysis.

Tocris products are intended for laboratory research use only, unless stated otherwise.

Product Datasheets for CHEB

Certificate of Analysis is currently unavailable on-line.
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References for CHEB

References are publications that support the biological activity of the product.

Nicholson et al (1990) Calcium-dependent actions of the convulsant barbiturate, CHEB, on transmitter release at the rat neuromuscular junction. Gen.Pharmacol. 21 741 PMID: 1980474

Wei et al (1996) CHEB, a convulsant barbiturate, evokes calcium dependant spontaneous glutamate release from rat cerebrocortical synaptosomes. Neuropharmacology 35 695 PMID: 8887978

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Keywords: CHEB, CHEB supplier, GABAA, Receptors, 0311, Tocris Bioscience

Citations for CHEB

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Currently there are no citations for CHEB.

Reviews for CHEB

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Literature in this Area

Tocris offers the following scientific literature in this area to showcase our products. We invite you to request* your copy today!

*Please note that Tocris will only send literature to established scientific business / institute addresses.


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GABA Receptors Scientific Review

GABA Receptors Scientific Review

Written by Ian Martin, Norman Bowery and Susan Dunn, this review provides a history of the GABA receptor, as well as discussing the structure and function of the various subtypes and the clinical potential of receptor modulators; compounds available from Tocris are listed.

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Epilepsy is a brain disease that affects 60 million people globally. More than 20 anti-seizure drugs are currently available, but these do not address the underlying causes of the condition. This poster summarizes current knowledge about the development of the condition and highlights some approaches that have disease-modifying effects in proof-of-concept studies.

Pain Poster

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Peripheral sensitization is the reduction in the threshold of excitability of sensory neurons that results in an augmented response to a given external stimulus. This poster outlines the excitatory and inhibitory signaling pathways involved in modulation of peripheral sensitization. The role of ion channels, GPCRs, neurotrophins, and cytokines in sensory neurons are also described.

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Schizophrenia is a debilitating psychiatric disorder that affects 1% of the worldwide population. This poster describes the neurobiology of Schizophrenia, as well as highlighting the genetic and environmental factors that play a fundamental role in the etiology of the disease. The current and emerging drug targets are also discussed.