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Discontinued ProductEtonitazenyl isothiocyanate (Cat. No. 0516) has been withdrawn from sale for commercial reasons.
Biological Activity for Etonitazenyl isothiocyanate
Affinity label specific for μ opioid sites.
Technical Data for Etonitazenyl isothiocyanate
|Storage||Desiccate at -20°C|
The technical data provided above is for guidance only. For batch specific data refer to the Certificate of Analysis.
Tocris products are intended for laboratory research use only, unless stated otherwise.
Product Datasheets for Etonitazenyl isothiocyanate
References for Etonitazenyl isothiocyanate
References are publications that support the biological activity of the product.
Burke et al (1984) Probes for narcotic receptor mediated phenomena. 7. Synthesis and pharmacological properties of irreversible ligands specific for μ or δ opiate receptors. J.Med.Chem. 27 1570 PMID: 6150112
Keywords: Etonitazenyl isothiocyanate, Etonitazenyl isothiocyanate supplier, Irreversible, affinity, label, μ-opioid, mu-opioid, selective, MOP, Receptors, OP3, BIT, Mu, Opioid, 0516, Tocris Bioscience
Citations for Etonitazenyl isothiocyanate
Citations are publications that use Tocris products.
Currently there are no citations for Etonitazenyl isothiocyanate.
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Literature in this Area
Tocris offers the following scientific literature in this area to showcase our products. We invite you to request* your copy today!
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Peptides Involved in Appetite Modulation Scientific Review
Written by Sonia Tucci, Lynsay Kobelis and Tim Kirkham, this review provides a synopsis of the increasing number of peptides that have been implicated in appetite regulation and energy homeostasis; putative roles of the major peptides are outlined and compounds available from Tocris are listed.
The key feature of drug addiction is the inability to stop using a drug despite clear evidence of harm. This poster describes the brain circuits associated with addiction, and provides an overview of the main classes of addictive drugs and the neurotransmitter systems that they target.
Peripheral sensitization is the reduction in the threshold of excitability of sensory neurons that results in an augmented response to a given external stimulus. This poster outlines the excitatory and inhibitory signaling pathways involved in modulation of peripheral sensitization. The role of ion channels, GPCRs, neurotrophins, and cytokines in sensory neurons are also described.