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Biological Activity for Carboprost tromethamine
Carboprost tromethamine is a synthetic analog of prostaglandin F2α (PGF2α, Cat. No. 4214). Increases uterine contractility in the 14-day pregnant hamster.
Technical Data for Carboprost tromethamine
|Storage||Store at -20°C|
The technical data provided above is for guidance only. For batch specific data refer to the Certificate of Analysis.
Tocris products are intended for laboratory research use only, unless stated otherwise.
Solubility Data for Carboprost tromethamine
|Solvent||Max Conc. mg/mL||Max Conc. mM|
Preparing Stock Solutions for Carboprost tromethamine
The following data is based on the product molecular weight 489.64. Batch specific molecular weights may vary from batch to batch due to the degree of hydration, which will affect the solvent volumes required to prepare stock solutions.
|Concentration / Solvent Volume / Mass||1 mg||5 mg||10 mg|
|1 mM||2.04 mL||10.21 mL||20.42 mL|
|5 mM||0.41 mL||2.04 mL||4.08 mL|
|10 mM||0.2 mL||1.02 mL||2.04 mL|
|50 mM||0.04 mL||0.2 mL||0.41 mL|
Product Datasheets for Carboprost tromethamine
References for Carboprost tromethamine
References are publications that support the biological activity of the product.
Vickery et al (1979) Opposite and mutually antagonistic effects on uterine contractility of two epimeric forms of a synthetic prostaglandin analog. Prostaglandins Med. 2 3 PMID: 550139
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Keywords: Carboprost tromethamine, Carboprost tromethamine supplier, prostaglandin, F2alpha, analogs, PGF2alpha, receptor, agonists, carboprost, trometamol, U32921, Prostanoid, Receptors, 6428, Tocris Bioscience
Citations for Carboprost tromethamine
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Literature in this Area
Tocris offers the following scientific literature in this area to showcase our products. We invite you to request* your copy today!
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Peripheral sensitization is the reduction in the threshold of excitability of sensory neurons that results in an augmented response to a given external stimulus. This poster outlines the excitatory and inhibitory signaling pathways involved in modulation of peripheral sensitization. The role of ion channels, GPCRs, neurotrophins, and cytokines in sensory neurons are also described.