Amyloid β-peptide (25-35) (human)

Pricing Availability   Qty
Description: Human amyloid β-protein fragment functionally required for neurotoxicity
Purity: ≥95% (HPLC)
Citations (1)
Literature (1)

Biological Activity for Amyloid β-peptide (25-35) (human)

Amyloid β-peptide (25-35) (human) is a fragment of human amyloid β-peptide, functionally required for the neurotrophic and neurotoxic effects associated with Alzheimer's disease.

Technical Data for Amyloid β-peptide (25-35) (human)

M. Wt 1060.27
Formula C45H81N13O14S
Storage Store at -20°C
Purity ≥95% (HPLC)
CAS Number 131602-53-4
PubChem ID 10843733
Smiles [H]NCC(=O)N[C@@H](CO)C(=O)N[C@@H](CC(N)=O)C(=O)N[C@@H](CCCCN)C(=O)NCC(=O)N[C@@H](C)C(=O)N[C@@H]([C@@H](C)CC)C(=O)N[C@@H]([C@@H](C)CC)C(=O)NCC(=O)N[C@@H](CC(C)C)C(=O)N[C@@H](CCSC)C(O)=O

The technical data provided above is for guidance only. For batch specific data refer to the Certificate of Analysis.

Tocris products are intended for laboratory research use only, unless stated otherwise.

Solubility Data for Amyloid β-peptide (25-35) (human)

Solubility Soluble to 0.50 mg/ml in water

Product Datasheets for Amyloid β-peptide (25-35) (human)

Certificate of Analysis / Product Datasheet
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References for Amyloid β-peptide (25-35) (human)

References are publications that support the biological activity of the product.

Rush et al (1992) Intracerebral β-amyloid(25-35) produces tissue damage: is it neurotoxic? Neurobiol.Aging 13 591 PMID: 1281289

Schenk et al (1995) Therapeutic approaches related to amyloid-β peptide and Alzheimer's Disease J.Med.Chem. 38 4141 PMID: 7473539

Yankner et al (1990) Neurotrophic and neurotoxic effects of amyoid β protein: reversal by tachykinin neuropeptides. Science 250 279 PMID: 2218531

If you know of a relevant reference for Amyloid β-peptide (25-35) (human), please let us know.

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1 Citation for Amyloid β-peptide (25-35) (human)

Citations are publications that use Tocris products. Selected citations for Amyloid β-peptide (25-35) (human) include:

Ostrovskaya et al (2014) Neuroprotective effect of novel cognitive enhancer noopept on AD-related cellular model involves the attenuation of apoptosis and tau hyperphosphorylation. J.Biomed.Sci. 21 74 PMID: 25096780

Do you know of a great paper that uses Amyloid β-peptide (25-35) (human) from Tocris? Please let us know.

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Literature in this Area

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Alzheimer's Disease Poster

Alzheimer's Disease Poster

Alzheimer's disease (AD) is a debilitating and progressive neurodegenerative disease and the most common cause of dementia, affecting approximately 30% of individuals aged over 85 years. This poster summarizes the cellular and molecular mechanisms of AD.