Discontinued ProductUnfortunately 3-Hydroxy-2-quinoxalinecarboxylic acid (Cat. No. 0184) has been withdrawn from sale for commercial reasons.
Broad spectrum antagonist at ionotropic glutamate receptors.
|Storage||Store at RT|
The technical data provided above is for guidance only. For batch specific data refer to the Certificate of Analysis.
All Tocris products are intended for laboratory research use only.
Preparing Stock Solutions
The following data is based on the product molecular weight 190.16. Batch specific molecular weights may vary from batch to batch due to solvent of hydration, which will affect the solvent volumes required to prepare stock solutions.
|Concentration / Solvent Volume / Mass||1 mg||5 mg||10 mg|
|1 mM||5.26 mL||26.29 mL||52.59 mL|
|5 mM||1.05 mL||5.26 mL||10.52 mL|
|10 mM||0.53 mL||2.63 mL||5.26 mL|
|50 mM||0.11 mL||0.53 mL||1.05 mL|
References are publications that support the products' biological activity.
Erez et al (1985) Anticonvulsant properties of 3-hydroxy-2-quinoxalinecarboxylic acid, a newly found antagonist of excitatory amino acids. Eur.J.Pharmacol. 110 31 PMID: 2861099
Frey et al (1988) 6,7-Dichloro-3-hydroxy-2-quinoxalinecarboxylic acid is a relatively potent antagonist at NMDA and kainate receptors. Neurosci.Lett. 91 194 PMID: 2847085
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Keywords: 3-Hydroxy-2-quinoxalinecarboxylic acid, supplier, Non-selective, Ionotropic, Glutamate, Non-selective, Ionotropic, Glutamate, Tocris Bioscience
Citations for 3-Hydroxy-2-quinoxalinecarboxylic acid
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Literature in this Area
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Peripheral sensitization is the reduction in the threshold of excitability of sensory neurons that results in an augmented response to a given external stimulus. This poster outlines the excitatory and inhibitory signaling pathways involved in modulation of peripheral sensitization. The role of ion channels, GPCRs, neurotrophins, and cytokines in sensory neurons are also described.
Parkinson's disease (PD) causes chronic disability and is the second most common neurodegenerative condition. This poster outlines the neurobiology of the disease, as well as highlighting current therapeutic treatments for symptomatic PD, and emerging therapeutic strategies to delay PD onset and progression.