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Products (128)
Targets (5)
Resources (5)

"apoptosis" has 128 results in Products.

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Apoptosis Activator 2

Cat. No. 2098/10
Description: Promotes apoptosome formation and activates caspase-9/caspase-3 pathway. Selectively induces tumor cell apoptosis
Purity: ≥99% (HPLC)
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2,3-DCPE hydrochloride

Cat. No. 2137/10
Description: Selectively induces cancer cell apoptosis
Purity: ≥99% (HPLC)
Pricing & Availability Qty

iMAC2

Cat. No. 3794/10
Description: Suppressor of mitochondrial apoptosis
Purity: ≥98% (HPLC)
Pricing & Availability Qty

UC 112

Cat. No. 5251/10
Description: IAP and XIAP inhibitor; antitumor
Purity: ≥98% (HPLC)
Pricing & Availability Qty

2-Methoxyestradiol

Cat. No. 1807/10
Description: Apoptotic and antiangiogenic agent
Purity: ≥99% (HPLC)
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CHM 1

Cat. No. 3868/10
Description: Potent antitumor agent; inducer of apoptosis
Purity: ≥99% (HPLC)
Pricing & Availability Qty

Carboplatin

Cat. No. 2626/50
Description: DNA cross-linking antitumor agent
Purity: ≥99% (HPLC)
Pricing & Availability Qty

Deguelin

Cat. No. 1770/10
Description: Anticancer and antiviral agent; chemopreventive and pro-apoptotic
Purity: ≥97% (HPLC)
Pricing & Availability Qty

Streptozocin

Cat. No. 1621/100
Description: DNA alkylator; antitumor and induces diabetes
Purity: ≥95% (HPLC)
Pricing & Availability Qty

YM 155

Cat. No. 6491/10
Description: Survivin suppressor
Purity: ≥98% (HPLC)
Pricing & Availability Qty
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"apoptosis" has 5 results in Targets.

ASK1

Apoptosis signal-regulated kinase 1 (ASK1) is a serine/threonine kinase, MAP3K family member, which induces apoptosis through the activation of JNK and p38. ASK1 has been implicated in the pathology of neurodegenerative and oxidative stress-related diseases.

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Apoptosis

Apoptosis is a mechanism of cell death that occurs after sufficient cellular damage. It occurs normally during development and aging and is considered a vital component of cell turnover, cell development and function of the immune system. It occurs as a defense mechanism such as in immune reactions or when cells are damaged by disease or noxious agents.

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Apoptosis Inducers

Apoptosis inducers exhibit pro-apoptotic effects through a variety of mechanisms, including DNA cross-linking, inhibition of antiapoptotic proteins and activation of caspases. These inducers may target a specific cellular process in order to induce antitumor or antineoplastic effects.

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Inhibitor of Apoptosis

The inhibitor of apoptosis (IAP) family of proteins are major regulators of apoptosis through interaction with components of the apoptotic signaling cascades. Discovered in baculovirus, human homologs can be described as baculovirus IAP repeat containing (BIRC) proteins.

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"apoptosis" has 5 results in Resources.

Apoptosis Signaling Pathway

Apoptosis is a physiological process for cell death that is critical during aging and development. It may also be referred to as cell 'suicide'. Apoptosis can be triggered by events both inside and outside of the cell.

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Cancer Research Product Guide

A collection of over 750 products for cancer research, the guide includes research tools for the study of:

  • Cancer Metabolism
  • Epigenetics in Cancer
  • Receptor Signaling
  • Cell Cycle and DNA Damage Repair
  • Angiogenesis
  • Invasion and Metastasis
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Huntington's Disease Life Science Poster

Huntington's disease (HD) is a monogenic neurodegenerative disorder, which is characterized by the prevalent loss of GABAergic medium spiny neurons (MSN) in the striatum. This poster summarizes the MSN intracellular signaling pathways implicated in the pathology of HD, as well as highlighting the use of iPSCs for HD modeling.

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Neurodegeneration Research Product Guide

A collection of over 275 products for neurodegeneration research, the guide includes research tools for the study of:

  • Alzheimer's disease
  • Parkinson's disease
  • Huntington's disease
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Programmed Cell Death Life Science Poster

There are two currently recognized forms of programmed cell death: apoptosis and necroptosis. This poster summarizes the signaling pathways involved in apoptosis, necroptosis and cell survival following death receptor activation, and highlights the influence of the molecular switch, cFLIP, on cell fate.

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